阻断Notch信号的角质形成细胞对成纤维细胞增殖及分泌功能的影响研究(4)
[7]Nowinski D,Hoijer P,Engstrand T,et al.Keratinocytes inhibit expression of
connective tissue growth factor in fibroblasts in vivo by an interleukin-lα-dq)endent mechanism[J]J Invest Dermatol,2002,l 19(2):449-455.
[8]Rangarajan A,Talora C,Okuyama R,et al.Notch signaling is adirect determinant of keratinocyte growth arrest and entry into
differentiation[J].EMBO J,2001,20(13):3427-3436.
, http://www.100md.com
[9]Dotto GRNotch tumor suppressor function[J].Oncogene,2008,27(38):5115-5123.
[10]夏煒,潘宝华,刘宾,等.Notch信号在增生性瘢痕表皮中的表达[J].中华整形外科杂志,2009,25(1):41-45.
[11]Li Bing,Guo Shuzhong,Xia Wei,et al.Blocking Notch signal pathwaysuppress profibrotic growth factor production in keratinocyte [J].Chinese JAesthet Med,2012,21(4):576-579.
[12]刁建升,张曦,夏炜,等.阻断Notch信号对兔耳增生性瘢痕形成的影响及作用机制[J].中华整形外科杂志,2009,89(16):1088-1092.
, 百拇医药
[13]Lowell S,Jones P,Le Roux I,et al.Stimulation of human epidermaldifferentiation by Deltα-Notch signaling at the boundaries of stem-cell clusters [J].Curr Biol,2000,10(9):491-500.
[14]Ryubei Okuyama,Hachiro Tagami,Setsuya Aiba.Notch signaling:Its role in epidermal homeostasis and in pathogenesis of skindiseases[J].J Dermatol Sci,2008,49(3): 187-194.
[15]Lim IJ,Phan TT,Song C,et al.Investigation of the influence ofkeloid-derived keratinocyte on fibroblast growth and proliferation invitro[J].Plast Reconstr Surg,2001,107(3):797-808.
, 百拇医药
[16]Shi C,Zhu J,Yang D.The pivotal role of inflammation in scar/keloidformation after acne[J].Dermatoendocrinol,2018,9(l):el448327.
[17]Leavitt T,Hu MS,Marshall CD,et al.Scarless wound healingrfindingthe right cells and signals[J].Cell Tissue Res,2016,365(3):483-493.
[18]Ma X,Chen J,Xu B,et aLKeloid-derived keratinocytes acquire a fibroblast-like appearance and an enhanced invasive capacity in a hypoxicmicroenvironment in vitro[J].Int J Mol Med,2015,35(5): 1246-1256.
, 百拇医药
[19]Jiao Y,Wang X,Zhang J,et al.Inhibiting function of human fetal dermalmesenchymal stem cells on bioactivities of keloid fibroblasts [J].StemCell Res Ther,2017,8(1):170.
[20]Wu N,Rollin J,Masse I,et al.p63 regulates human keratinocyteproliferation via MYC-regulated gene network and differentiationcommitment through cell adhesion-related gene network[J].J BiolChem,2012,287(8):5627-5638.
[21]Dong S,Sun Y.MicroRNΑ-22 may promote apoptosis and inhibit theproliferation of hypertrophic scar fibroblasts by regulating the mitogen-activated protein kinase kinase/extracellular signal-regulated kinase/p21pathway [J].Exp Ther Med,2017,14(4):3841-3845.
, 百拇医药
[22]Luo L,Li J,Liu H,et al.Adiponectin is involved in connective tissuegrowth factor-induced proliferation,migration and overproduction of theextracellular matrix in keloid fibroblasts[J].Int J Moi Sci,2017,18(5):el044.
[23]Wang X,Cao P,Liu J,et al.5-Aminolaevulinic acid-based photodynamictherapy restrains pathological hyperplasia of fibroblasts [J], Med SciMonit,2017,23:46-56.
[24]Jiao H,Dong P,Yan L,et al.TGF-pi induces polypyrimidine tract-binding protein to alter fibroblasts proliferation and fibronectindeposition in keloid[J].Sci Rep,2016,6:38033.
, 百拇医药
[25]Wang W,Qu M,Xu L,et al.Sorafenib exerts an anti-keloid activity byantagonizing TGF-p/Smad and MAPK/ERK signaling pathways[J].JMol Med(Berl).2016,94(10): 1181-1194.
[26]Farhatullah S,Ardeshir B.Notch signaling pathway in keloid disease:Enhanced fibroblast activity in a Jagged-1 peptide-dependentmanner in lesional vs.extralesional fibroblasts[J].Wound RepairRegen,2012,20(5):688-706., 百拇医药(楚菲菲 王大雷 闫伦 李辉超 夏炜 马显杰)
connective tissue growth factor in fibroblasts in vivo by an interleukin-lα-dq)endent mechanism[J]J Invest Dermatol,2002,l 19(2):449-455.
[8]Rangarajan A,Talora C,Okuyama R,et al.Notch signaling is adirect determinant of keratinocyte growth arrest and entry into
differentiation[J].EMBO J,2001,20(13):3427-3436.
, http://www.100md.com
[9]Dotto GRNotch tumor suppressor function[J].Oncogene,2008,27(38):5115-5123.
[10]夏煒,潘宝华,刘宾,等.Notch信号在增生性瘢痕表皮中的表达[J].中华整形外科杂志,2009,25(1):41-45.
[11]Li Bing,Guo Shuzhong,Xia Wei,et al.Blocking Notch signal pathwaysuppress profibrotic growth factor production in keratinocyte [J].Chinese JAesthet Med,2012,21(4):576-579.
[12]刁建升,张曦,夏炜,等.阻断Notch信号对兔耳增生性瘢痕形成的影响及作用机制[J].中华整形外科杂志,2009,89(16):1088-1092.
, 百拇医药
[13]Lowell S,Jones P,Le Roux I,et al.Stimulation of human epidermaldifferentiation by Deltα-Notch signaling at the boundaries of stem-cell clusters [J].Curr Biol,2000,10(9):491-500.
[14]Ryubei Okuyama,Hachiro Tagami,Setsuya Aiba.Notch signaling:Its role in epidermal homeostasis and in pathogenesis of skindiseases[J].J Dermatol Sci,2008,49(3): 187-194.
[15]Lim IJ,Phan TT,Song C,et al.Investigation of the influence ofkeloid-derived keratinocyte on fibroblast growth and proliferation invitro[J].Plast Reconstr Surg,2001,107(3):797-808.
, 百拇医药
[16]Shi C,Zhu J,Yang D.The pivotal role of inflammation in scar/keloidformation after acne[J].Dermatoendocrinol,2018,9(l):el448327.
[17]Leavitt T,Hu MS,Marshall CD,et al.Scarless wound healingrfindingthe right cells and signals[J].Cell Tissue Res,2016,365(3):483-493.
[18]Ma X,Chen J,Xu B,et aLKeloid-derived keratinocytes acquire a fibroblast-like appearance and an enhanced invasive capacity in a hypoxicmicroenvironment in vitro[J].Int J Mol Med,2015,35(5): 1246-1256.
, 百拇医药
[19]Jiao Y,Wang X,Zhang J,et al.Inhibiting function of human fetal dermalmesenchymal stem cells on bioactivities of keloid fibroblasts [J].StemCell Res Ther,2017,8(1):170.
[20]Wu N,Rollin J,Masse I,et al.p63 regulates human keratinocyteproliferation via MYC-regulated gene network and differentiationcommitment through cell adhesion-related gene network[J].J BiolChem,2012,287(8):5627-5638.
[21]Dong S,Sun Y.MicroRNΑ-22 may promote apoptosis and inhibit theproliferation of hypertrophic scar fibroblasts by regulating the mitogen-activated protein kinase kinase/extracellular signal-regulated kinase/p21pathway [J].Exp Ther Med,2017,14(4):3841-3845.
, 百拇医药
[22]Luo L,Li J,Liu H,et al.Adiponectin is involved in connective tissuegrowth factor-induced proliferation,migration and overproduction of theextracellular matrix in keloid fibroblasts[J].Int J Moi Sci,2017,18(5):el044.
[23]Wang X,Cao P,Liu J,et al.5-Aminolaevulinic acid-based photodynamictherapy restrains pathological hyperplasia of fibroblasts [J], Med SciMonit,2017,23:46-56.
[24]Jiao H,Dong P,Yan L,et al.TGF-pi induces polypyrimidine tract-binding protein to alter fibroblasts proliferation and fibronectindeposition in keloid[J].Sci Rep,2016,6:38033.
, 百拇医药
[25]Wang W,Qu M,Xu L,et al.Sorafenib exerts an anti-keloid activity byantagonizing TGF-p/Smad and MAPK/ERK signaling pathways[J].JMol Med(Berl).2016,94(10): 1181-1194.
[26]Farhatullah S,Ardeshir B.Notch signaling pathway in keloid disease:Enhanced fibroblast activity in a Jagged-1 peptide-dependentmanner in lesional vs.extralesional fibroblasts[J].Wound RepairRegen,2012,20(5):688-706., 百拇医药(楚菲菲 王大雷 闫伦 李辉超 夏炜 马显杰)